In the past 12 months there have been many interesting publications on paralytic strabismus. Many investigators have reported on various imaging techniques helping both the clinician and the basic scientist better evaluate the causes and effects of paralytic strabismus on the extraocular muscle environment. Some interesting cases of neurologic and systemic associations have also been reported, including a unilateral pupil-sparing third nerve paralysis developing after the use of sildenafil citrate (Viagra; Pfizer, New York, New York). Advances in genetics are now helping us further understand complex entities such as progressive external ophthalmoparesis and congenital fibrosis syndrome. At the other end of the spectrum, colleagues are reporting clinical findings to help specify the diagnosis of conditions as diverse as degenerative ataxic disorders and masked bilateral superior oblique muscle paresis. Unfortunately, there were very few papers on proven new effective surgical techniques for the treatment of paralytic strabismus this year. Finally, to help clarify terminology, and when at all possible depending on the reports reviewed, the following nomenclature will be used in this paper: paralysis--complete loss of function of a muscle or group of muscles innervated by a specified (when applicable) cranial nerve or branch; paresis--incomplete loss of function; and palsy--when the authors failed to specify the degree of weakness.