Abstract
Although hypercoagulable states are most often associated with venous thrombosis, arterial thromboses are reported in protein S, protein C, and antithrombin III deficiencies, factor V Leiden and prothrombin gene mutations, hyperhomocysteinemia, dysfibrinogenemia, plasminogen deficiency, sickle cell disease, and antiphospholipid antibody syndrome.
MeSH terms
-
Anemia, Sickle Cell / etiology
-
Antiphospholipid Syndrome / immunology
-
Antiphospholipid Syndrome / physiopathology
-
Blood Protein Disorders / classification
-
Blood Protein Disorders / complications
-
Blood Protein Disorders / genetics
-
Blood Protein Disorders / metabolism
-
Disseminated Intravascular Coagulation / complications*
-
Factor V / genetics
-
Factor V / metabolism
-
Fibrinogens, Abnormal / genetics
-
Fibrinogens, Abnormal / metabolism
-
Humans
-
Hyperhomocysteinemia / etiology
-
Hyperhomocysteinemia / genetics
-
Plasminogen / genetics
-
Plasminogen / metabolism
-
Prothrombin / genetics
-
Prothrombin / metabolism
-
Stroke / etiology
-
Stroke / genetics
-
Stroke / metabolism*
Substances
-
Fibrinogens, Abnormal
-
factor V Leiden
-
Factor V
-
Prothrombin
-
Plasminogen