Coagulopathies and arterial stroke

J Neuroophthalmol. 2003 Mar;23(1):63-71. doi: 10.1097/00041327-200303000-00012.

Abstract

Although hypercoagulable states are most often associated with venous thrombosis, arterial thromboses are reported in protein S, protein C, and antithrombin III deficiencies, factor V Leiden and prothrombin gene mutations, hyperhomocysteinemia, dysfibrinogenemia, plasminogen deficiency, sickle cell disease, and antiphospholipid antibody syndrome.

Publication types

  • Review

MeSH terms

  • Anemia, Sickle Cell / etiology
  • Antiphospholipid Syndrome / immunology
  • Antiphospholipid Syndrome / physiopathology
  • Blood Protein Disorders / classification
  • Blood Protein Disorders / complications
  • Blood Protein Disorders / genetics
  • Blood Protein Disorders / metabolism
  • Disseminated Intravascular Coagulation / complications*
  • Factor V / genetics
  • Factor V / metabolism
  • Fibrinogens, Abnormal / genetics
  • Fibrinogens, Abnormal / metabolism
  • Humans
  • Hyperhomocysteinemia / etiology
  • Hyperhomocysteinemia / genetics
  • Plasminogen / genetics
  • Plasminogen / metabolism
  • Prothrombin / genetics
  • Prothrombin / metabolism
  • Stroke / etiology
  • Stroke / genetics
  • Stroke / metabolism*

Substances

  • Fibrinogens, Abnormal
  • factor V Leiden
  • Factor V
  • Prothrombin
  • Plasminogen