Diagnostic difficulties with common SURF1 mutations in patients with cytochrome oxidase-deficient Leigh syndrome

J Inherit Metab Dis. 2004;27(1):57-65. doi: 10.1023/B:BOLI.0000016622.05609.b8.

Abstract

In three unrelated patients with systemic cytochrome oxidase deficiency resulting from mutations in the SURF1 gene, the same mutation in the splice donor site of intron 3 was identified. All three patients were compound heterozygotes, two for the common insertion/deletion mutation in exon 4. In all three cases, complete definition of the causative mutations was only resolved by combined analysis of cDNA and genomic DNA. Several factors were identified that contributed to the diagnostic difficulties: preferential amplification of deleted cDNA, significant formation of heteroduplexes in cDNA PCR amplification and unequal representation of heterozygous peaks in genomic DNA sequences. These patients emphasize the need to perform mutation analysis on both cDNA and genomic DNA wherever possible.

MeSH terms

  • Alternative Splicing
  • Base Sequence
  • Cells, Cultured
  • Cytochrome-c Oxidase Deficiency / diagnosis*
  • Cytochrome-c Oxidase Deficiency / genetics*
  • DNA Mutational Analysis
  • DNA Transposable Elements
  • DNA, Complementary
  • Diagnosis, Differential
  • Exons
  • Gene Amplification
  • Gene Deletion
  • Heterozygote
  • Humans
  • Introns
  • Leigh Disease / diagnosis*
  • Leigh Disease / genetics*
  • Membrane Proteins
  • Mitochondrial Proteins
  • Molecular Sequence Data
  • Mutation*
  • Nucleic Acid Heteroduplexes
  • Polymerase Chain Reaction
  • Proteins / genetics*
  • RNA Splice Sites

Substances

  • DNA Transposable Elements
  • DNA, Complementary
  • Membrane Proteins
  • Mitochondrial Proteins
  • Nucleic Acid Heteroduplexes
  • Proteins
  • RNA Splice Sites
  • Surf-1 protein