[FIP1L1-PDGFRalpha-positive myeloproliferative disease with hypereosinophilia: clinical characteristics and pathogenetic therapy]

Ter Arkh. 2005;77(7):90-2.
[Article in Russian]
No abstract available

Publication types

  • Case Reports
  • Comparative Study

MeSH terms

  • Adult
  • Antineoplastic Agents / therapeutic use
  • Benzamides
  • DNA, Neoplasm / genetics
  • Disease Progression
  • Female
  • Follow-Up Studies
  • Gene Expression Regulation, Neoplastic
  • Humans
  • Hypereosinophilic Syndrome / drug therapy
  • Hypereosinophilic Syndrome / etiology*
  • Hypereosinophilic Syndrome / genetics
  • Imatinib Mesylate
  • Karyotyping
  • Male
  • Myeloproliferative Disorders / complications
  • Myeloproliferative Disorders / drug therapy
  • Myeloproliferative Disorders / genetics*
  • Oncogene Proteins, Fusion
  • Piperazines / therapeutic use
  • Point Mutation
  • Pyrimidines / therapeutic use
  • Receptor, Platelet-Derived Growth Factor alpha / genetics*
  • Reverse Transcriptase Polymerase Chain Reaction
  • mRNA Cleavage and Polyadenylation Factors / genetics*

Substances

  • Antineoplastic Agents
  • Benzamides
  • DNA, Neoplasm
  • Oncogene Proteins, Fusion
  • Piperazines
  • Pyrimidines
  • mRNA Cleavage and Polyadenylation Factors
  • Imatinib Mesylate
  • FIP1L1-PDGFRA fusion protein, human
  • Receptor, Platelet-Derived Growth Factor alpha