Mutations of N-RAS, FLT3 and p53 genes are not involved in the development of acute leukemia transformed from myeloproliferative diseases with JAK2 mutation

Leukemia. 2006 Jun;20(6):1168-9. doi: 10.1038/sj.leu.2404186.
No abstract available

Publication types

  • Letter

MeSH terms

  • Acute Disease
  • Adult
  • Aged
  • Aged, 80 and over
  • Female
  • Humans
  • Janus Kinase 2
  • Leukemia, Myeloid / genetics*
  • Male
  • Middle Aged
  • Mutation
  • Myeloproliferative Disorders / genetics*
  • Precursor Cell Lymphoblastic Leukemia-Lymphoma / genetics*
  • Protein-Tyrosine Kinases / genetics*
  • Proto-Oncogene Proteins / genetics*
  • Proto-Oncogene Proteins p21(ras) / genetics*
  • Tumor Suppressor Protein p53 / genetics*
  • fms-Like Tyrosine Kinase 3 / genetics*

Substances

  • Proto-Oncogene Proteins
  • Tumor Suppressor Protein p53
  • FLT3 protein, human
  • Protein-Tyrosine Kinases
  • fms-Like Tyrosine Kinase 3
  • JAK2 protein, human
  • Janus Kinase 2
  • Proto-Oncogene Proteins p21(ras)