Sodium-hydrogen exchanger, cardiac overload, and myocardial hypertrophy

Circulation. 2007 Mar 6;115(9):1090-100. doi: 10.1161/CIRCULATIONAHA.106.626929.
No abstract available

Publication types

  • Research Support, Non-U.S. Gov't
  • Review

MeSH terms

  • Amiloride / pharmacology
  • Amiloride / therapeutic use
  • Angiotensin II / physiology
  • Animals
  • Calcium Signaling
  • Carbonic Anhydrase II / physiology
  • Cardiomegaly / physiopathology*
  • Cardiomegaly / prevention & control
  • Cation Transport Proteins / antagonists & inhibitors
  • Cation Transport Proteins / chemistry
  • Cation Transport Proteins / physiology*
  • Cells, Cultured / drug effects
  • Cells, Cultured / metabolism
  • Endothelins / physiology
  • Heart Failure / drug therapy
  • Heart Failure / etiology
  • Heart Failure / physiopathology
  • Hormones / physiology
  • Humans
  • Hydrogen / metabolism
  • Hydrogen-Ion Concentration
  • Hypertrophy, Left Ventricular / etiology
  • Hypertrophy, Left Ventricular / physiopathology
  • Hypertrophy, Left Ventricular / prevention & control
  • MAP Kinase Signaling System
  • Mice
  • Mitochondria, Heart / drug effects
  • Models, Cardiovascular
  • Myocytes, Cardiac / drug effects
  • Myocytes, Cardiac / metabolism
  • Phosphorylation
  • Protein Processing, Post-Translational
  • Rabbits
  • Rats
  • Rats, Inbred SHR
  • Reactive Oxygen Species
  • Signal Transduction
  • Sodium / metabolism
  • Sodium-Hydrogen Exchanger 1
  • Sodium-Hydrogen Exchangers / antagonists & inhibitors
  • Sodium-Hydrogen Exchangers / chemistry
  • Sodium-Hydrogen Exchangers / physiology*
  • Stress, Mechanical
  • Swine

Substances

  • Cation Transport Proteins
  • Endothelins
  • Hormones
  • Reactive Oxygen Species
  • SLC9A1 protein, human
  • Sodium-Hydrogen Exchanger 1
  • Sodium-Hydrogen Exchangers
  • Angiotensin II
  • Amiloride
  • Hydrogen
  • Sodium
  • Carbonic Anhydrase II