Germline mutations in SPI1 and MADD do not contribute to familial chronic lymphocytic leukaemia

Leukemia. 2007 Jun;21(6):1315-8. doi: 10.1038/sj.leu.2404646. Epub 2007 Apr 5.
No abstract available

Publication types

  • Letter
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Death Domain Receptor Signaling Adaptor Proteins
  • Family Health
  • Female
  • Germ-Line Mutation
  • Guanine Nucleotide Exchange Factors / genetics*
  • Humans
  • Leukemia, Lymphocytic, Chronic, B-Cell / etiology*
  • Leukemia, Lymphocytic, Chronic, B-Cell / genetics
  • Male
  • Middle Aged
  • Proto-Oncogene Proteins / genetics*
  • Trans-Activators / genetics*

Substances

  • Death Domain Receptor Signaling Adaptor Proteins
  • Guanine Nucleotide Exchange Factors
  • MADD protein, human
  • Proto-Oncogene Proteins
  • Trans-Activators
  • proto-oncogene protein Spi-1