Peroxisome proliferator-activated receptor gamma and early folliculogenesis during an acute hyperandrogenism condition

Fertil Steril. 2011 Jan;95(1):333-7. doi: 10.1016/j.fertnstert.2010.07.1083.

Abstract

Acute hyperandrogenism decreases serum P levels and induces early apoptosis of antral follicles by a mechanism mediated by the peroxisome proliferator-activated receptor gamma system and independent of the steroidogenic acute regulator protein.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acute Disease
  • Animals
  • Apoptosis / drug effects
  • Apoptosis / physiology
  • Chorionic Gonadotropin / pharmacology*
  • Dehydroepiandrosterone / pharmacology
  • Female
  • Gene Expression / drug effects
  • Hyperandrogenism / metabolism
  • Hyperandrogenism / pathology
  • Hyperandrogenism / physiopathology*
  • Ovarian Follicle / drug effects*
  • Ovarian Follicle / pathology
  • Ovarian Follicle / physiology*
  • PPAR gamma / genetics
  • PPAR gamma / metabolism*
  • Phosphoproteins / genetics
  • Phosphoproteins / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Reproductive Control Agents / pharmacology

Substances

  • Chorionic Gonadotropin
  • PPAR gamma
  • Phosphoproteins
  • Reproductive Control Agents
  • steroidogenic acute regulatory protein
  • Dehydroepiandrosterone