Abstract
In this issue of Blood, Poulain et al demonstrate the high prevalence of the MYD88 L265P somatic mutation in patients with Waldenstrom macroglobulinemia (WM) and provide insight into its biological relevance in the growth and survival of WM.
Publication types
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Research Support, Non-U.S. Gov't
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Comment
MeSH terms
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Female
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Humans
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Male
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Myeloid Differentiation Factor 88 / genetics*
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Point Mutation*
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Waldenstrom Macroglobulinemia / genetics*
Substances
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MYD88 protein, human
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Myeloid Differentiation Factor 88