Abstract
In this issue of Blood, Treon and colleagues provide strong evidence that mutations in MYD88 and CXCR4 dictate clinical presentation and survival in Waldenström macroglobulinemia (WM).
MeSH terms
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Female
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Humans
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Male
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Mutation*
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Myeloid Differentiation Factor 88 / genetics*
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Receptors, CXCR4 / genetics*
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Waldenstrom Macroglobulinemia / genetics*
Substances
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Myeloid Differentiation Factor 88
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Receptors, CXCR4