Abstract
Genetic inactivation of PRC2 components, NF1, and CDKN2A are frequently detected in MPNSTs.
©2014 American Association for Cancer Research.
MeSH terms
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Cell Line, Tumor
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Cyclin-Dependent Kinase Inhibitor p16 / genetics
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Humans
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Mutation
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Neoplasm Proteins
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Nerve Sheath Neoplasms / genetics*
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Neurofibromin 1 / genetics
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Polycomb Repressive Complex 2 / genetics*
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Transcription Factors
Substances
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Cyclin-Dependent Kinase Inhibitor p16
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Neoplasm Proteins
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Neurofibromin 1
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SUZ12 protein, human
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Transcription Factors
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Polycomb Repressive Complex 2