Mutual exclusivity between the fusion gene CBFB::MYH11 and somatic mitochondrial mutations in acute myeloid leukaemia

Br J Haematol. 2022 Nov;199(4):e25-e29. doi: 10.1111/bjh.18444. Epub 2022 Aug 29.

Authors

Chaoxian Zhao^{1

2}, Siyang Wang^{1

2

3}, Kankan Wang^{1

2

3}

Affiliations

¹ State Key Laboratory of Medical Genomics, Shanghai Institute of Hematology, Ruijin Hospital Affiliated to Shanghai Jiao Tong University School of Medicine, Shanghai, China.
² Sino-French Research Center for Life Sciences and Genomics, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China.
³ School of Life Sciences and Biotechnology, Shanghai Jiao Tong University, Shanghai, China.

PMID: 36039018
DOI: 10.1111/bjh.18444

No abstract available

Publication types

Letter
Research Support, Non-U.S. Gov't

MeSH terms

Chromosome Inversion
Core Binding Factor beta Subunit / genetics
DNA, Mitochondrial* / genetics
DNA, Mitochondrial* / metabolism
Humans
Leukemia, Myeloid, Acute* / genetics
Mitochondria / genetics
Mutation
Myosin Heavy Chains / genetics
Oncogene Proteins, Fusion / genetics

Substances

CBFB protein, human
Core Binding Factor beta Subunit
MYH11 protein, human
Myosin Heavy Chains
Oncogene Proteins, Fusion
DNA, Mitochondrial