YKL40 potentially via activating the P38 pathway in decreasing atherosclerotic plaque stability

Chin Med J (Engl). 2024 Apr 20;137(8):1003-1005. doi: 10.1097/CM9.0000000000003038. Epub 2024 Mar 26.

Authors

Chao Wang¹, Yandong Liu^{1

2}, Wei Huan¹, Jianjin Wu¹, Jie Jin¹, Sili Zou¹, Yu Chen³, Lefeng Qu¹

Affiliations

¹ Department of Vascular and Endovascular Surgery, Changzheng Hospital Affiliated to the Naval Medical University, Shanghai 200003, China.
² Department of Emergency, Navy 905th Hospital, Shanghai 200050, China.
³ Clinical Research Institute of Integrative Medicine, Yueyang Hospital, Shanghai University of Traditional Chinese Medicine, Shanghai 200437, China.

No abstract available

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Adipokines* / metabolism
Animals
Chitinase-3-Like Protein 1* / metabolism
Humans
Lectins / metabolism
Mice
Plaque, Atherosclerotic* / metabolism
Plaque, Atherosclerotic* / pathology
p38 Mitogen-Activated Protein Kinases / metabolism

Substances

Chitinase-3-Like Protein 1
Adipokines
p38 Mitogen-Activated Protein Kinases
Lectins
CHI3L1 protein, human